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  1. 1
    Identification and characterization of the Ipomoea nil RelA/SpoT Homologs (InRSHs) and potential directions of their transcriptional regulation

    Online-Aufsatz

  2. 2
    17β estradiol regulates adhesion molecule expression in mesangial cells during glomerulonephritis
    von Jog, Neelakshi R
    Veröffentlicht in: Clinical immunology (Orlando, Fla.) (2015)

    Online-Aufsatz

  3. 3
    Study on the nuclear translocation mechanism in the inhibition of nuclear factor-ΚB activation in bacterial lipoprotein-tolerant THP-1 cells
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  4. 4
    Influence of inhibitory KappaB protein kinase alpha and gamma genes silencing induced by RNA interference on nuclear factor-KappaB signal pathway
    Aufsatz

  5. 5
    Oxidized lipids enhance RANKL production by T lymphocytes implications for lipid-induced bone loss
    von Graham, Lucia S
    Veröffentlicht in: Clinical immunology (Orlando, Fla.) (2009)

    Online-Aufsatz

  6. 6
    Primary investigation of the relationship between glucocorticoid induced leucine zipper and inflammatory reaction
    Aufsatz

  7. 7
    Fas (CD95) ligation inhibits activation of NF-kappa B by targeting p65-Rel A in a caspase-dependent manner
    von Wong, Henry K
    Veröffentlicht in: Clinical immunology (Orlando, Fla.) (2006)
    Aufsatz

  8. 8
    Two-step cross-linking method for identification of NF-kappaB gene network by chromatin immunoprecipitation
    von Nowak, David E
    Veröffentlicht in: BioTechniques (2005)
    Aufsatz

  9. 9
    Localized expressing tendency of nuclear transcription factor kappa-B, pro-fibrosis genetic factors and fibronectin mRNA in renal tissues in proteinuria overload nephrotic young rats
    Aufsatz

  10. 10
    Relativity of nuclear factor-kappaB (P65/Rel-A) and angiotensin II type 1 receptor expression in early stage of lesions of adriamycin nephrosis in young rats and the effects of intervention
    Aufsatz

  11. 11
    Regulation of NF-kappaB/P65 by MDM2 in acute lymphoblastic leukemia in childhood
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  12. 12
    Direct transfer of p65 into T lymphocytes from systemic lupus erythematosus patients leads to increased levels of interleukin-2 promoter activity
    Aufsatz

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