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231223s1997 xx ||||| 00| ||eng c |
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|a (NLM)15012275
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|a DE-627
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|e rakwb
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|a eng
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| 100 |
1 |
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|a Hammond-Kosack, Kim E.
|e verfasserin
|4 aut
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| 245 |
1 |
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|a PLANT DISEASE RESISTANCE GENES
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| 264 |
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|c 1997
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| 336 |
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|a Text
|b txt
|2 rdacontent
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| 337 |
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|a ohne Hilfsmittel zu benutzen
|b n
|2 rdamedia
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| 338 |
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|a Band
|b nc
|2 rdacarrier
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| 500 |
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|a Date Revised 09.01.2024
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| 500 |
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|a published: Print
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| 500 |
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|a Citation Status Publisher
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| 520 |
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|a In "gene-for-gene" interactions between plants and their pathogens, incompatibility (no disease) requires a dominant or semidominant resistance (R) gene in the plant, and a corresponding avirulence (Avr) gene in the pathogen. Many plant/pathogen interactions are of this type. R genes are presumed to (a) enable plants to detect Avr-gene-specified pathogen molecules, (b) initiate signal transduction to activate defenses, and (c) have the capacity to evolve new R gene specificities rapidly. Isolation of R genes has revealed four main classes of R gene sequences whose products appear to activate a similar range of defense mechanisms. Discovery of the structure of R genes and R gene loci provides insight into R gene function and evolution, and should lead to novel strategies for disease control
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| 650 |
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|a Journal Article
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| 700 |
1 |
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|a Jones, Jonathan D. G.
|e verfasserin
|4 aut
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| 773 |
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|i Enthalten in
|t Annual review of plant physiology and plant molecular biology
|d 1990
|g 48(1997) vom: 17. Juni, Seite 575-607
|w (DE-627)NLM098188720
|x 1040-2519
|7 nnns
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| 773 |
1 |
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|g volume:48
|g year:1997
|g day:17
|g month:06
|g pages:575-607
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|d 48
|j 1997
|b 17
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|h 575-607
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