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  1. 1
    JNK and phosphorylated Bcl-2 predict multiple sclerosis clinical activity and glatiramer acetate therapeutic response

    Article en ligne

  2. 2
    Biological activity of glatiramer acetate on Treg and anti-inflammatory monocytes persists for more than 10years in responder multiple sclerosis patients

    Article en ligne

  3. 3
    Effector and regulatory B cells in Multiple Sclerosis

    Article en ligne

  4. 4
    Identification of a gene expression signature in peripheral blood of multiple sclerosis patients treated with disease-modifying therapies

    Article en ligne

  5. 5
    The low EOMES/TBX21 molecular phenotype in multiple sclerosis reflects CD56+ cell dysregulation and is affected by immunomodulatory therapies

    Article en ligne

  6. 6
    Modulation of immune function occurs within hours of therapy initiation for multiple sclerosis

    Article en ligne

  7. 7
    Epitope-specific immune tolerization ameliorates experimental autoimmune encephalomyelitis

    Article en ligne

  8. 8
    Failure of glatiramer acetate to modify the peripheral T cell repertoire of relapsing-remitting multiple sclerosis patients

    Article en ligne

  9. 9
    CD4(+)CD25(+) regulatory T cells contribute to the therapeutic effects of glatiramer acetate in experimental autoimmune encephalomyelitis
    par Jee, Y
    Publié dans: Clinical immunology (Orlando, Fla.) (2007)
    Article

  10. 10
    Amelioration of experimental colitis by Copaxone is associated with class-II-restricted CD4 immune blocking
    Article

  11. 11
    Inflammatory potential and migratory capacities across human brain endothelial cells of distinct glatiramer acetate-reactive T cells generated in treated multiple sclerosis patients
    Article

  12. 12
    In vitro evidence that subcutaneous administration of glatiramer acetate induces hyporesponsive T cells in patients with multiple sclerosis
    Article

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