Myocardia-Injected Synergistically Anti-Apoptotic and Anti-Inflammatory Poly(amino acid) Hydrogel Relieves Ischemia-Reperfusion Injury

© 2025 Wiley‐VCH GmbH.

Bibliographische Detailangaben
Veröffentlicht in:Advanced materials (Deerfield Beach, Fla.). - 1998. - 37(2025), 11 vom: 31. März, Seite e2420171
1. Verfasser: Luo, Qiang (VerfasserIn)
Weitere Verfasser: Li, Zhibo, Sun, Wei, Wang, Guoliang, Yao, Haochen, Wang, Guoqing, Liu, Bin, Ding, Jianxun
Format: Online-Aufsatz
Sprache:English
Veröffentlicht: 2025
Zugriff auf das übergeordnete Werk:Advanced materials (Deerfield Beach, Fla.)
Schlagworte:Journal Article in situ myocardial injection anti‐apoptosis anti‐inflammation ischemia‐reperfusion injury therapy poly(amino acid) hydrogel Hydrogels Anti-Inflammatory Agents Fingolimod Hydrochloride G926EC510T mehr... Reactive Oxygen Species Polyethylene Glycols 3WJQ0SDW1A Amino Acids
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520 |a Reperfusion therapy is the most effective treatment for acute myocardial infarction, but its efficacy is frequently limited by ischemia-reperfusion injury (IRI). While antioxidant and anti-inflammatory therapies have shown significant potential in alleviating IRI, these strategies have not yielded satisfactory clinical outcomes. For that, a thermo-sensitive myocardial-injectable poly(amino acid) hydrogel of methoxy poly(ethylene glycol)45-poly(L-methionine20-co-L-alanine10) (mPEG45-P(Met20-co-Ala10), PMA) loaded with FTY720 (PMA/FTY720) is developed to address IRI through synergistic anti-apoptotic and anti-inflammatory effects. Upon injection into the ischemic myocardium, the PMA aqueous solution undergoes a sol-to-gel phase transition and gradually degrades in response to reactive oxygen species (ROS), releasing FTY720 on demand. PMA acts synergistically with FTY720 to inhibit cardiomyocyte apoptosis and modulate pro-inflammatory M1 macrophage polarization toward anti-inflammatory M2 macrophages by clearing ROS, thereby mitigating the inflammatory response and promoting vascular regeneration. In a rat IRI model, PMA/FTY720 reduces the apoptotic cell ratio by 81.8%, increases vascular density by 34.0%, and enhances left ventricular ejection fraction (LVEF) by 12.8%. In a rabbit IRI model, the gel-based sustained release of FTY720 enhanced LVEF by an additional 7.2% compared to individual treatment. In summary, the engineered PMA hydrogel effectively alleviates IRI through synergistic anti-apoptosis and anti-inflammation actions, offering valuable clinical potential for treating myocardial IRI 
650 4 |a Journal Article 
650 4 |a in situ myocardial injection 
650 4 |a anti‐apoptosis 
650 4 |a anti‐inflammation 
650 4 |a ischemia‐reperfusion injury therapy 
650 4 |a poly(amino acid) hydrogel 
650 7 |a Hydrogels  |2 NLM 
650 7 |a Anti-Inflammatory Agents  |2 NLM 
650 7 |a Fingolimod Hydrochloride  |2 NLM 
650 7 |a G926EC510T  |2 NLM 
650 7 |a Reactive Oxygen Species  |2 NLM 
650 7 |a Polyethylene Glycols  |2 NLM 
650 7 |a 3WJQ0SDW1A  |2 NLM 
650 7 |a Amino Acids  |2 NLM 
700 1 |a Li, Zhibo  |e verfasserin  |4 aut 
700 1 |a Sun, Wei  |e verfasserin  |4 aut 
700 1 |a Wang, Guoliang  |e verfasserin  |4 aut 
700 1 |a Yao, Haochen  |e verfasserin  |4 aut 
700 1 |a Wang, Guoqing  |e verfasserin  |4 aut 
700 1 |a Liu, Bin  |e verfasserin  |4 aut 
700 1 |a Ding, Jianxun  |e verfasserin  |4 aut 
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773 1 8 |g volume:37  |g year:2025  |g number:11  |g day:31  |g month:03  |g pages:e2420171 
856 4 0 |u http://dx.doi.org/10.1002/adma.202420171  |3 Volltext 
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