Protein phosphatase StTOPP6 negatively regulates potato bacterial wilt resistance by modulating MAPK signaling

© The Author(s) 2023. Published by Oxford University Press on behalf of the Society for Experimental Biology. All rights reserved. For permissions, please email: journals.permissionsoup.com.

Bibliographische Detailangaben
Veröffentlicht in:Journal of experimental botany. - 1985. - 74(2023), 14 vom: 03. Aug., Seite 4208-4224
1. Verfasser: Wang, Bingsen (VerfasserIn)
Weitere Verfasser: Huang, Mengshu, He, Wenfeng, Wang, Yuqi, Yu, Liu, Zhou, Dan, Meng, Chengzhen, Cheng, Dong, Qiu, Huishan, Tan, Xiaodan, Song, Botao, Chen, Huilan
Format: Online-Aufsatz
Sprache:English
Veröffentlicht: 2023
Zugriff auf das übergeordnete Werk:Journal of experimental botany
Schlagworte:Journal Article Research Support, Non-U.S. Gov't Ralstonia solanacearum Solanum tuberosum Bacterial wilt MAPK StTOPP6 disease resistance potato protein phosphatase mehr... Mitogen-Activated Protein Kinases EC 2.7.11.24 Reactive Oxygen Species Phosphoprotein Phosphatases EC 3.1.3.16
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520 |a © The Author(s) 2023. Published by Oxford University Press on behalf of the Society for Experimental Biology. All rights reserved. For permissions, please email: journals.permissionsoup.com. 
520 |a Potato (Solanum tuberosum) is an important crop globally and is grown across many regions in China, where it ranks fourth in the list of staple foods. However, its production and quality are severely affected by bacterial wilt caused by Ralstonia solanacearum. In this study, we identified StTOPP6, which belongs to the type one protein phosphatase (TOPP) family, and found that transient knock down of StTOPP6 in potato increased resistance against R. solanacearum. RNA-seq analysis showed that knock down of StTOPP6 activated immune responses, and this defense activation partly depended on the mitogen-activated protein kinase (MAPK) signal pathway. StTOPP6 inhibited the expression of StMAPK3, while overexpression of StMAPK3 enhanced resistance to R. solanacearum, supporting the negative role of StTOPP6 in plant immunity. Consistent with the results of knock down of StTOPP6, overexpressing the phosphatase-dead mutation StTOPP6m also attenuated infection and up-regulated MAPK3, showing that StTOPP6 activity is required for disease. Furthermore, we found that StTOPP6 affected the StMAPK3-mediated downstream defense pathway, eventually suppressing the accumulation of reactive oxygen species (ROS). Consistent with these findings, plants with knock down of StTOPP6, overexpression of StTOPP6m, and overexpression of StMAPK3 all displayed ROS accumulation and enhanced resistance to R. solanacearum. Taken together, the findings of our study demonstrate that StTOPP6 negatively regulates resistance to bacterial wilt by affecting the MAPK3-mediated pathway 
650 4 |a Journal Article 
650 4 |a Research Support, Non-U.S. Gov't 
650 4 |a Ralstonia solanacearum 
650 4 |a Solanum tuberosum 
650 4 |a Bacterial wilt 
650 4 |a MAPK 
650 4 |a StTOPP6 
650 4 |a disease resistance 
650 4 |a potato 
650 4 |a protein phosphatase 
650 7 |a Mitogen-Activated Protein Kinases  |2 NLM 
650 7 |a EC 2.7.11.24  |2 NLM 
650 7 |a Reactive Oxygen Species  |2 NLM 
650 7 |a Phosphoprotein Phosphatases  |2 NLM 
650 7 |a EC 3.1.3.16  |2 NLM 
700 1 |a Huang, Mengshu  |e verfasserin  |4 aut 
700 1 |a He, Wenfeng  |e verfasserin  |4 aut 
700 1 |a Wang, Yuqi  |e verfasserin  |4 aut 
700 1 |a Yu, Liu  |e verfasserin  |4 aut 
700 1 |a Zhou, Dan  |e verfasserin  |4 aut 
700 1 |a Meng, Chengzhen  |e verfasserin  |4 aut 
700 1 |a Cheng, Dong  |e verfasserin  |4 aut 
700 1 |a Qiu, Huishan  |e verfasserin  |4 aut 
700 1 |a Tan, Xiaodan  |e verfasserin  |4 aut 
700 1 |a Song, Botao  |e verfasserin  |4 aut 
700 1 |a Chen, Huilan  |e verfasserin  |4 aut 
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