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231225s2018 xx |||||o 00| ||eng c |
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|a 10.1016/j.jplph.2018.09.006
|2 doi
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|a pubmed24n0962.xml
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|a (NLM)30253266
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|a (PII)S0176-1617(18)30157-3
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|a DE-627
|b ger
|c DE-627
|e rakwb
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|a eng
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|a Salazar-Iribe, Alexis
|e verfasserin
|4 aut
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|a BIIDXI, a DUF642 cell wall protein, is involved in hypocotyl growth via auxin efflux
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|c 2018
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|a Text
|b txt
|2 rdacontent
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|a ƒaComputermedien
|b c
|2 rdamedia
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|a ƒa Online-Ressource
|b cr
|2 rdacarrier
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|a Date Completed 17.01.2019
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|a Date Revised 30.09.2020
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|a published: Print-Electronic
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|a Citation Status MEDLINE
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|a Copyright © 2018 Elsevier GmbH. All rights reserved.
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|a Auxin is involved in hypocotyl elongation in response to different environmental factors. BIIDXI is a cell wall DUF642 protein that participates in the regulation of the degree of pectin-methylesterification of the cell wall in different tissues, including hypocotyls. Under continuous light, bdx-1 seedlings presented longer hypocotyls than those of WT, while BIIDXI-overexpressed hypocotyls were auxin resistant. Auxin accumulation was observed in epidermal cells from bdx-1 hypocotyls, and the distribution pattern of PIN1 proteins differed. Moreover, the gravitropic response of bdx-1, a process that is highly dependent on auxin flux, was increased. In this study, we determined that BIIDXI is involved in hypocotyl elongation through the regulation of auxin flux
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|a Journal Article
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|a Auxin
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|a DUF642 cell wall proteins
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| 650 |
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|a Hypocotyl elongation
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| 650 |
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|a Arabidopsis Proteins
|2 NLM
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| 650 |
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|a BIIDXI protein, Arabidopsis
|2 NLM
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| 650 |
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|a Carrier Proteins
|2 NLM
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| 650 |
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|a Indoleacetic Acids
|2 NLM
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| 650 |
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|a Plant Growth Regulators
|2 NLM
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1 |
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|a Cruz-Valderrama, José Erik
|e verfasserin
|4 aut
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1 |
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|a Jímenez-Durán, Karina
|e verfasserin
|4 aut
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| 700 |
1 |
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|a Gómez-Maqueo, Ximena
|e verfasserin
|4 aut
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| 700 |
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|a Gamboa-deBuen, Alicia
|e verfasserin
|4 aut
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| 773 |
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|i Enthalten in
|t Journal of plant physiology
|d 1979
|g 231(2018) vom: 01. Dez., Seite 105-109
|w (DE-627)NLM098174622
|x 1618-1328
|7 nnns
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| 773 |
1 |
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|g volume:231
|g year:2018
|g day:01
|g month:12
|g pages:105-109
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|u http://dx.doi.org/10.1016/j.jplph.2018.09.006
|3 Volltext
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