Study of an extended family with CTLA-4 deficiency suggests a CD28/CTLA-4 independent mechanism responsible for differences in disease manifestations and severity

Study of an extended family with CTLA-4 deficiency suggests a CD28/CTLA-4 independent mechanism responsible for differences in disease manifestations and severity

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Détails bibliographiques
Publié dans:Clinical immunology (Orlando, Fla.). - 1999. - 188(2018) vom: 01. März, Seite 94-102
Auteur principal: Hou, Tie Zheng (Auteur)
Autres auteurs: Olbrich, Peter, Soto, Jose Manuel Lucena, Sanchez, Berta, Moreno, Paula Sanchez, Borte, Stephan, Stauss, Hans J, Burns, Siobhan O, Walker, Lucy S K, Pan-Hammarström, Qiang, Hammarström, Lennart, Sansom, David M, Neth, Olaf
Format: Article en ligne
Langue:English
Publié: 2018
Accès à la collection:Clinical immunology (Orlando, Fla.)
Sujets:Journal Article Research Support, Non-U.S. Gov't Autoimmunity CD28 CTLA-4 Immunodeficiency Mutation Regulatory T cells CD28 Antigens CTLA-4 Antigen CTLA4 protein, human
Accès en ligne Volltext