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NLM273303090 |
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231225s2017 xx |||||o 00| ||eng c |
| 024 |
7 |
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|a 10.1016/j.jsha.2016.11.001
|2 doi
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| 028 |
5 |
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|a pubmed24n0911.xml
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|a eng
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| 100 |
1 |
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|a Boussir, Hanane
|e verfasserin
|4 aut
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| 245 |
1 |
0 |
|a Eosinophilic myocarditis and hypereosinophilic syndrome
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| 264 |
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1 |
|c 2017
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| 336 |
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|a Text
|b txt
|2 rdacontent
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| 337 |
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|a ƒaComputermedien
|b c
|2 rdamedia
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| 338 |
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|a ƒa Online-Ressource
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|2 rdacarrier
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| 500 |
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|a Date Revised 30.09.2020
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|a published: Print-Electronic
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|a Citation Status PubMed-not-MEDLINE
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|a Hypereosinophilic syndrome (HES) is a heterogeneous group of hematological disorders characterized by a chronic, unexplained hypereosinophilia with tissue damage. Cardiac involvement occurs in ∼20% of patients with HES and represents a major turning point. Cardiac injuries related to eosinophilia are divided into three chronological phases: eosinophilic infiltration, thrombosis, and fibrosis. We report a case of a 33-year-old woman diagnosed with HES, with pulmonary and gastrointestinal involvement and eosinophilic myocarditis in cardiogenic shock. The evolution was favorable with dobutamine, anticoagulation, corticosteroids, and later, β-blockers and angiotensin-converting enzyme inhibitors. Cardiac involvement in HES is rare but carries a poor prognosis. Corticosteroids are considered by many to be the mainstay of treatment. Although new treatments have been suggested, only a few seem promising
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| 650 |
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|a Journal Article
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| 650 |
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|a Cardiogenic shock
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| 650 |
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4 |
|a Corticosteroids
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| 650 |
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4 |
|a Eosinophilic myocarditis
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| 650 |
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4 |
|a Hypereosinophilic syndrome
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| 650 |
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4 |
|a Imatinib
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| 700 |
1 |
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|a Ghalem, Amine
|e verfasserin
|4 aut
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| 700 |
1 |
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|a Ismaili, Nabila
|e verfasserin
|4 aut
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| 700 |
1 |
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|a El Ouafi, Noha
|e verfasserin
|4 aut
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| 773 |
0 |
8 |
|i Enthalten in
|t Journal of the Saudi Heart Association
|d 1999
|g 29(2017), 3 vom: 14. Juli, Seite 211-213
|w (DE-627)NLM098225227
|x 1016-7315
|7 nnns
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| 773 |
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|g volume:29
|g year:2017
|g number:3
|g day:14
|g month:07
|g pages:211-213
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4 |
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|u http://dx.doi.org/10.1016/j.jsha.2016.11.001
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|h 211-213
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