PKA Inhibitor H89 (N-[2-p-bromocinnamylamino-ethyl]-5-isoquinolinesulfonamide) Attenuates Synaptic Dysfunction and Neuronal Cell Death following Ischemic Injury
The cyclic AMP-dependent protein kinase (PKA), which activates prosurvival signaling proteins, has been implicated in the expression of long-term potentiation and hippocampal long-term memory. It has come to light that H89 commonly known as the PKA inhibitor have diverse roles in the nervous system...
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Bibliographische Detailangaben
| Veröffentlicht in: | Neural plasticity. - 1998. - 2015(2015) vom: 01., Seite 374520
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| 1. Verfasser: |
Song, Juhyun
(VerfasserIn) |
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| Weitere Verfasser: |
Cheon, So Yeong,
Lee, Won Taek,
Park, Kyung Ah,
Lee, Jong Eun |
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| Format: | Online-Aufsatz
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| Sprache: | English |
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| Veröffentlicht: |
2015
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| Zugriff auf das übergeordnete Werk: | Neural plasticity
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| Schlagworte: | Journal Article
Research Support, Non-U.S. Gov't
Apoptosis Regulatory Proteins
Brain-Derived Neurotrophic Factor
Disks Large Homolog 4 Protein
Dlg4 protein, mouse
Isoquinolines
Membrane Proteins
Microtubule-Associated Proteins
Mtap2 protein, mouse
mehr...
Protein Kinase Inhibitors
Sulfonamides
Cyclic AMP-Dependent Protein Kinases
EC 2.7.11.11
Guanylate Kinases
EC 2.7.4.8
N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide
M876330O56 |
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