Enhanced activity of galactono-1,4-lactone dehydrogenase and ascorbate-glutathione cycle in mitochondria from complex III deficient Arabidopsis

Copyright © 2011 Elsevier Masson SAS. All rights reserved.

Bibliographische Detailangaben
Veröffentlicht in:Plant physiology and biochemistry : PPB. - 1991. - 49(2011), 8 vom: 15. Aug., Seite 809-15
1. Verfasser: Zsigmond, Laura (VerfasserIn)
Weitere Verfasser: Tomasskovics, Bálint, Deák, Veronika, Rigó, Gábor, Szabados, László, Bánhegyi, Gábor, Szarka, András
Format: Online-Aufsatz
Sprache:English
Veröffentlicht: 2011
Zugriff auf das übergeordnete Werk:Plant physiology and biochemistry : PPB
Schlagworte:Journal Article Research Support, Non-U.S. Gov't Antioxidants Arabidopsis Proteins pentatricopeptide repeat protein, Arabidopsis Ascorbate Peroxidases EC 1.11.1.11 Oxidoreductases Acting on CH-CH Group Donors EC 1.3.- galactonolactone dehydrogenase mehr... EC 1.3.2.3 NADH, NADPH Oxidoreductases EC 1.6.- monodehydroascorbate reductase (NADH) EC 1.6.5.4 Glutathione Reductase EC 1.8.1.7 Electron Transport Complex III EC 7.1.1.8 Glutathione GAN16C9B8O Ascorbic Acid PQ6CK8PD0R Dehydroascorbic Acid Y2Z3ZTP9UM
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245 1 0 |a Enhanced activity of galactono-1,4-lactone dehydrogenase and ascorbate-glutathione cycle in mitochondria from complex III deficient Arabidopsis 
264 1 |c 2011 
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520 |a Copyright © 2011 Elsevier Masson SAS. All rights reserved. 
520 |a The mitochondrial antioxidant homeostasis was investigated in Arabidopsis ppr40-1 mutant, which presents a block of electron flow at complex III. The activity of the ascorbate biosynthetic enzyme, L-galactono-1,4-lactone dehydrogenase (EC 1.3.2.3) (GLDH) was elevated in mitochondria isolated from mutant plants. In addition increased activities of the enzymes of Foyer-Halliwell-Asada cycle and elevated glutathione (GSH) level were observed in the mutant mitochondria. Lower ascorbate and ascorbate plus dehydroascorbate contents were detected at both cellular and mitochondrial level. Moreover, the more oxidized mitochondrial redox status of ascorbate in the ppr40-1 mutant indicated that neither the enhanced activity of GLDH nor Foyer-Halliwell-Asada cycle could compensate for the enhanced ascorbate consumption in the absence of a functional respiratory chain 
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650 7 |a galactonolactone dehydrogenase  |2 NLM 
650 7 |a EC 1.3.2.3  |2 NLM 
650 7 |a NADH, NADPH Oxidoreductases  |2 NLM 
650 7 |a EC 1.6.-  |2 NLM 
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700 1 |a Tomasskovics, Bálint  |e verfasserin  |4 aut 
700 1 |a Deák, Veronika  |e verfasserin  |4 aut 
700 1 |a Rigó, Gábor  |e verfasserin  |4 aut 
700 1 |a Szabados, László  |e verfasserin  |4 aut 
700 1 |a Bánhegyi, Gábor  |e verfasserin  |4 aut 
700 1 |a Szarka, András  |e verfasserin  |4 aut 
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