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231223s2011 xx |||||o 00| ||eng c |
| 024 |
7 |
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|a 10.1111/j.1365-2761.2011.01237.x
|2 doi
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|a eng
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| 100 |
1 |
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|a Tinsley, J W
|e verfasserin
|4 aut
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| 245 |
1 |
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|a Novel non-motile phenotypes of Yersinia ruckeri suggest expansion of the current clonal complex theory
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| 264 |
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|c 2011
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| 336 |
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|a Text
|b txt
|2 rdacontent
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| 337 |
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|a ƒaComputermedien
|b c
|2 rdamedia
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| 338 |
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|a ƒa Online-Ressource
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| 500 |
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|a Date Completed 13.07.2011
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|a Date Revised 30.09.2020
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|a published: Print-Electronic
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|a Citation Status MEDLINE
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| 520 |
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|a © 2011 Blackwell Publishing Ltd.
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|a The biochemical and cell surface characteristics of 63 non-motile isolates of Yersinia ruckeri from various sources were compared using the API 20E rapid identification system and conventional phenotypic methods. Eight individual phenotypic groups from a variety of fish species were observed from the data set. Non-motile isolates were not exclusively observed from serogroup O1; membership of biotype 2 was recorded for representatives from serogroups O2-O7. Variations in phenotypes highlights that new clonal groups are arising and that the current typing scheme requires expansion. Previously, it was hypothesized that disease was caused by a few virulent clones; data in this paper suggests that this assumption is not the case. The lipopolysaccharide (O antigen) type in the non-motile biotype was different from other isolates of Y. ruckeri
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| 650 |
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|a Journal Article
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| 650 |
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7 |
|a Bacterial Outer Membrane Proteins
|2 NLM
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| 650 |
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7 |
|a O Antigens
|2 NLM
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| 700 |
1 |
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|a Austin, D A
|e verfasserin
|4 aut
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| 700 |
1 |
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|a Lyndon, A R
|e verfasserin
|4 aut
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| 700 |
1 |
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|a Austin, B
|e verfasserin
|4 aut
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| 773 |
0 |
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|i Enthalten in
|t Journal of fish diseases
|d 1998
|g 34(2011), 4 vom: 01. Apr., Seite 311-7
|w (DE-627)NLM098166034
|x 1365-2761
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| 773 |
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|g volume:34
|g year:2011
|g number:4
|g day:01
|g month:04
|g pages:311-7
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|u http://dx.doi.org/10.1111/j.1365-2761.2011.01237.x
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