Oral sulforaphane increases Phase II antioxidant enzymes in the human upper airway
BACKGROUND: Cellular oxidative stress is an important factor in asthma and is thought to be the principle mechanism by which oxidant pollutants such as ozone and particulates mediate their pro-inflammatory effects. Endogenous Phase II enzymes abrogate oxidative stress through the scavenging of react...
Ausführliche Beschreibung
Bibliographische Detailangaben
| Veröffentlicht in: | Clinical immunology (Orlando, Fla.). - 1999. - 130(2009), 3 vom: 15. März, Seite 244-51
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| 1. Verfasser: |
Riedl, Marc A
(VerfasserIn) |
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| Weitere Verfasser: |
Saxon, Andrew,
Diaz-Sanchez, David |
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| Format: | Online-Aufsatz
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| Sprache: | English |
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| Veröffentlicht: |
2009
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| Zugriff auf das übergeordnete Werk: | Clinical immunology (Orlando, Fla.)
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| Schlagworte: | Clinical Trial
Journal Article
Research Support, N.I.H., Extramural
Antioxidants
Enzymes
Glucosinolates
Isothiocyanates
Sulfoxides
Thiocyanates
isothiocyanic acid
mehr...
3129-90-6
Heme Oxygenase-1
EC 1.14.14.18
NAD(P)H Dehydrogenase (Quinone)
EC 1.6.5.2
Glutathione Transferase
EC 2.5.1.18
sulforaphane
GA49J4310U |
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