Silencing of acidic pathogenesis-related PR-1 genes increases extracellular beta-(1->3)-glucanase activity at the onset of tobacco defence reactions

The class 1 pathogenesis-related (PR) proteins are thought to be involved in plant defence responses, but their molecular functions are unknown. The function of PR-1 was investigated in tobacco by generating stable PR-1a-silenced lines in which other acidic PR-1 genes (PR-1b and PR-1c) were silenced...

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Veröffentlicht in:Journal of experimental botany. - 1985. - 59(2008), 6 vom: 01., Seite 1225-39
1. Verfasser: Rivière, Marie-Pierre (VerfasserIn)
Weitere Verfasser: Marais, Antoine, Ponchet, Michel, Willats, William, Galiana, Eric
Format: Online-Aufsatz
Sprache:English
Veröffentlicht: 2008
Zugriff auf das übergeordnete Werk:Journal of experimental botany
Schlagworte:Journal Article Research Support, Non-U.S. Gov't Algal Proteins Fungal Proteins Glucans Plant Proteins Protein Isoforms Recombinant Proteins cryptogein protein, Phytophthora cryptogea pathogenesis-related proteins, plant mehr... callose 9064-51-1 Glucan 1,3-beta-Glucosidase EC 3.2.1.58 Salicylic Acid O414PZ4LPZ
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100 1 |a Rivière, Marie-Pierre  |e verfasserin  |4 aut 
245 1 0 |a Silencing of acidic pathogenesis-related PR-1 genes increases extracellular beta-(1->3)-glucanase activity at the onset of tobacco defence reactions 
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520 |a The class 1 pathogenesis-related (PR) proteins are thought to be involved in plant defence responses, but their molecular functions are unknown. The function of PR-1 was investigated in tobacco by generating stable PR-1a-silenced lines in which other acidic PR-1 genes (PR-1b and PR-1c) were silenced. Plants lacking extracellular PR-1s were more susceptible than wild-type plants to the oomycete Phytophthora parasitica but displayed unaffected systemic acquired resistance and developmental resistance to this pathogen. Treatment with salicylic acid up-regulates the PR-1g gene, encoding a basic protein of the PR-1 family, in PR-1-deficient tobacco, indicating that PR-1 expression may repress that of PR-1g. This shows that acidic PR-1s are dispensable for expression of salicylic acid-dependent acquired resistances against P. parasitica and may reveal a functional overlap in tobacco defence or a functional redundancy in the PR-1 gene family. The data also show that there is a specific increase in apoplastic beta-(1-->3)-glucanase activity and a decrease in beta-(1-->3)-glucan deposition in PR-1-silenced lines following activation of defence reactions. Complementation of the silencing by apoplastic treatment with a recombinant PR-1a protein largely restores the wild-type beta-(1-->3)-glucanase activity and callose phenotype. Taken together with the immunolocalization of PR-1a to sites of beta-(1-->3)-glucan deposition in wild-type plants, these results are indicative of a function for PR-1a in regulation of enzymatic activity of extracellular beta-(1-->3)-glucanases 
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650 7 |a Protein Isoforms  |2 NLM 
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650 7 |a cryptogein protein, Phytophthora cryptogea  |2 NLM 
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650 7 |a Salicylic Acid  |2 NLM 
650 7 |a O414PZ4LPZ  |2 NLM 
700 1 |a Marais, Antoine  |e verfasserin  |4 aut 
700 1 |a Ponchet, Michel  |e verfasserin  |4 aut 
700 1 |a Willats, William  |e verfasserin  |4 aut 
700 1 |a Galiana, Eric  |e verfasserin  |4 aut 
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