Activated sludge inhibition by chemical stressors--a comprehensive study
The effects of shock loads of 1-chloro-2,4-dinitrobenzene (CDNB); cadmium; 1-octanol; 2,4-dinitrophenol (DNP); weakly complexed cyanide; pH 5, 9, and 11; and high ammonia levels on activated sludge biomass growth, respiration rate, flocculation, chemical oxygen demand removal, dewaterability, and se...
Veröffentlicht in: | Water environment research : a research publication of the Water Environment Federation. - 1998. - 79(2007), 9 vom: 15. Sept., Seite 940-51 |
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Weitere Verfasser: | , , |
Format: | Aufsatz |
Sprache: | English |
Veröffentlicht: |
2007
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Zugriff auf das übergeordnete Werk: | Water environment research : a research publication of the Water Environment Federation |
Schlagworte: | Journal Article Research Support, Non-U.S. Gov't Cyanides Dinitrochlorobenzene Nitrites Water Pollutants, Chemical Cadmium 00BH33GNGH 1-Octanol NV1779205D mehr... |
Zusammenfassung: | The effects of shock loads of 1-chloro-2,4-dinitrobenzene (CDNB); cadmium; 1-octanol; 2,4-dinitrophenol (DNP); weakly complexed cyanide; pH 5, 9, and 11; and high ammonia levels on activated sludge biomass growth, respiration rate, flocculation, chemical oxygen demand removal, dewaterability, and settleability were studied. For all chemical shocks, except ammonia and pH, concentrations that caused 15, 25, and 50% respiration inhibition were used to provide a single pulse shock to sequencing batch reactor systems containing a nitrifying or non-nitrifying biomass. Cadmium and pH 11 shocks were most detrimental to all processes, followed by CDNB. The DNP and cyanide primarily affected respiration, while pH 5, pH 9, octanol, and ammonia did not affect the treatment process to a significant extent. A chemical source-process effect matrix is provided, which we believe will aid in the development of methods that prevent and/or attenuate the effects of toxic shock loads on activated sludge systems |
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Beschreibung: | Date Completed 04.01.2008 Date Revised 09.12.2020 published: Print Citation Status MEDLINE |
ISSN: | 1554-7531 |