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DE-627 |
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231223s2004 xx ||||| 00| ||eng c |
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|a DE-627
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|e rakwb
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|a eng
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| 100 |
1 |
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|a Williams, Jonathan
|e verfasserin
|4 aut
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| 245 |
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|a Dopamine appetite and cognitive impairment in attention deficit/hyperactivity disorder
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| 264 |
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|c 2004
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| 336 |
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|a Text
|b txt
|2 rdacontent
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| 337 |
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|a ohne Hilfsmittel zu benutzen
|b n
|2 rdamedia
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| 338 |
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|a Band
|b nc
|2 rdacarrier
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| 500 |
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|a Date Completed 07.09.2004
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|a Date Revised 16.03.2016
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|a published: Print
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|a Citation Status MEDLINE
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|a The underlying defects in ADHD (Attention Deficit/Hyperactivity Disorder) are not yet clear. The current paper tests three existing theories: State Regulation, Cognitive Deficit, and Temporal Difference (TD) learning. We present computational simulations of the Matching Familiar Figures Task and compare these with the experimental results reported by Sonuga-Barke (2002). The TD model contains four parameters: the learning rate, discounting for future rewards, brittleness (randomness) of behavior, and action bias. The results show that the basic TD model accounts well for control performance in trials of 5 sec, 10 sec, and 15 sec duration; but not for the deficits in ADHD performance at 5 sec and 15 sec. Extending the TD model to incorporate either a state regulation deficit, or working memory deficit and delay in starting trials, can provide a good account of both control and ADHD results, at all trial-lengths. We discuss the significance of the results for theories of ADHD and make suggestions for future experimentation
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| 650 |
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|a Comparative Study
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| 650 |
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|a Journal Article
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| 650 |
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|a Research Support, Non-U.S. Gov't
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| 650 |
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7 |
|a Dopamine
|2 NLM
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| 650 |
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7 |
|a VTD58H1Z2X
|2 NLM
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| 700 |
1 |
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|a Taylor, Eric
|e verfasserin
|4 aut
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| 773 |
0 |
8 |
|i Enthalten in
|t Neural plasticity
|d 1998
|g 11(2004), 1-2 vom: 06., Seite 115-32
|w (DE-627)NLM098558390
|x 1687-5443
|7 nnas
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| 773 |
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|g volume:11
|g year:2004
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|g pages:115-32
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|a AR
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| 952 |
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|d 11
|j 2004
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|h 115-32
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