Status Epilepticus Enhances Depotentiation after Fully Established LTP in an NMDAR-Dependent but GluN2B-Independent Manner

N-Methyl-D-aspartate (NMDA) receptor-dependent long-term potentiation (LTP) can be reversed by low-frequency stimulation (LFS) referred to as depotentiation (DP). We previously found GluN2B upregulated in CA1 neurons from post-status epilepticus (post-SE) tissue associated with an enhanced LTP. Here...

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Publié dans:	Neural plasticity. - 1998. - 2016(2016) vom: 15., Seite 6592038
Auteur principal:	Guli, Xiati (Auteur)
Autres auteurs:	Tokay, Tursonjan, Kirschstein, Timo, Köhling, Rüdiger
Format:	Article en ligne
Langue:	English
Publié:	2016
Accès à la collection:	Neural plasticity
Sujets:	Journal Article NR2B NMDA receptor Phenols Piperidines Receptors, N-Methyl-D-Aspartate Ro 25-6981 Pilocarpine 01MI4Q9DI3