Effects of nano-copper(II) oxide and nanomagnesium oxide particles on activated sludge
Effects of nano-copper(II) oxide (nano-CuO) and nanomagnesium oxide (nano-MgO) particles on activated sludge endogenous respiration (aerobic digestion), biochemical oxygen demand (BOD) biodegradation, and nitrification were investigated through respiration rate measurement. For comparison, the effec...
| Veröffentlicht in: | Water environment research : a research publication of the Water Environment Federation. - 1998. - 84(2012), 7 vom: 01. Juli, Seite 569-76 |
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| Format: | Aufsatz |
| Sprache: | English |
| Veröffentlicht: |
2012
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| Zugriff auf das übergeordnete Werk: | Water environment research : a research publication of the Water Environment Federation |
| Schlagworte: | Journal Article Research Support, U.S. Gov't, Non-P.H.S. Sewage Water Pollutants, Chemical Magnesium Oxide 3A3U0GI71G Copper 789U1901C5 cuprous oxide T8BEA5064F |
| Zusammenfassung: | Effects of nano-copper(II) oxide (nano-CuO) and nanomagnesium oxide (nano-MgO) particles on activated sludge endogenous respiration (aerobic digestion), biochemical oxygen demand (BOD) biodegradation, and nitrification were investigated through respiration rate measurement. For comparison, the effects of Cu(II) and Mg(II) ions on activated sludge were also studied. Results indicated that soluble Cu(II) has half maximum inhibitory concentration (IC50) values of 19, 5.5, 53, and 117 mg Cu/L for endogenous respiration, BOD biodegradation, ammonium oxidation, and nitrite oxidation, respectively. However, nano-CuO only inhibited BOD biodegradation at 240 mg Cu/L or more, and its associated toxicity was primarily caused by soluble Cu(II). In contrast, soluble Mg(II) was not toxic to activated sludge in the experimental concentration range, but nano-MgO inhibited BOD biodegradation and nitrification with IC50 values of 70 and 143 mg Mg/L, respectively. Further study indicated that the toxicity of nano-MgO resulted primarily from increased pH following MgO hydrolysis |
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| Beschreibung: | Date Completed 23.08.2012 Date Revised 23.09.2019 published: Print Citation Status MEDLINE |
| ISSN: | 1554-7531 |